Mitchell · Journal of sport rehabilitation 2009 · within-subject experimental physiological study · n=18

Neurophysiological reflex mechanisms' lack of contribution to the success of PNF stretches.

Level 3 - non-randomized controlled study

Non-randomized repeated-measures laboratory physiological study

PubMed 19827499 · doi:10.1123/jsr.18.3.343 · record verified 2026-08-26

What was done

Eighteen subjects aged 17–44 years performed two proprioceptive neuromuscular facilitation (PNF) stretching protocols: contract-relax (CR) and contract-relax, agonist contract (CRAC). Electromyographic (EMG) data were recorded from medial hamstring muscles using surface and indwelling wire electrodes to evaluate reciprocal inhibition, autogenic inhibition, and successive induction.

What was found

The abstract reports directional findings without numerical values. Reciprocal inhibition was not observed; instead, antagonist contraction produced elevated hamstring EMG activity suggestive of cocontraction. Autogenic inhibition was also not observed, with post-contraction EMG remaining higher than baseline. Successive induction was confirmed.

Why it matters

These findings challenge traditional explanations attributing the effectiveness of PNF stretching to spinal reflex inhibition mechanisms in healthy individuals.

Limits

The sample size is small (n = 18), and the abstract omits quantitative EMG data, statistical effect sizes, and range-of-motion measurements. Findings in healthy individuals may not generalize to populations with neurological impairment.

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